Prognosis Of Hsv Infection
HSV infections are often referred to in terms of the body part afflicted. For example, herpes that occur around the mouth and face are commonly known as or oral herpes; herpes that infect the genital tract are referred to as genital herpes. Although there are eight different strains, HSV1 and HSV2 are responsible for the majority of herpes infections. The cure for herpes symptoms is dependent on which strain of the virus is contracted.
Following active infection herpes viruses establish a latent infection in sensory and autonomic ganglia of the nervous system. The double-stranded DNA of the virus is incorporated into the cell physiology by infection of the nucleus of a nerve's cell body. HSV latency is static (no virus is produced) and is controlled by a number of viral genes, including Latency Associated Transcript (LAT).
Many HSV infected people experience recurrence within the first year of infection. Prodrome precedes development of lesions. Prodromal symptoms include tingling, itching, and pain where lumbosacral nerves innervate the skin. Prodrome may occur as long as several days or as short as a few hours before lesions develop. Beginning antiviral treatment when prodrome is experienced can reduce the appearance and duration of lesions in some individuals.
During recurrence fewer lesions are likely to develop, lesions are less painful and heal faster (within 5-10 days without antiviral treatment) than those occurring during the primary infection. Subsequent outbreaks tend to be periodic or episodic, occurring on average four to five times a year when not using antiviral therapy.
The causes of reactivation are uncertain, but several potential triggers have been documented. A recent study showed that a protein VP16 plays a key role in reactivation of the dormant virus. Changes in the immune system during menstruation may play a role in HSV-1 reactivation.
Concurrent infections, such as viral upper respiratory tract infection or other febrile diseases, can cause outbreaks. Reactivation due to infection is the likely source of the historic terms cold sore and fever blister. Other identified triggers include: local injury to the face, lips, eyes, or mouth; trauma; surgery; radiotherapy; and exposure to wind, ultraviolet light, or sunlight.
The frequency and severity of recurrent outbreaks vary greatly between patients. Some individuals' outbreaks can be quite debilitating with large, painful lesions persisting for several weeks, while others will experience only minor itching or burning for a few days. An immunity to the virus is built over time.
Most infected individuals will experience fewer outbreaks and outbreak symptoms will often become less severe. After several years, some people will become perpetually asymptomatic and will no longer experience outbreaks, though they may still be contagious to others. Immunocompromised individuals may experience episodes that are longer, more frequent, and more severe.
Antiviral medication has been proven to shorten the frequency and duration of outbreaks. Outbreaks may occur at the original site of the infection or in proximity to nerve endings that reach out from the infected ganglia. In the case of a genital infection, sores can appear at the original site of infection or near the base of the spine, the buttocks, or the back of the thighs.
The herpes virus remains dormant in the ganglia, seemingly inactive, however once provoked/aggravated, the virus becomes symptomatic. It travels down the ganglia and forms blisters on the surface of the skin causing an outbreak, often reoccurring in the same location.
As the pathogenic factors are highly symptomatic and acute during an outbreak, a high intensity treatment should be used during this stage, whereas a gentler antiviral treatment should be used when the virus is dormant, to help act as suppressive therapy, which is why we developed HSVSuppressor-Rx.
HSVSuppressor-Rx is a softer antiviral treatment designed for daily application/ suppressive therapy. It stays in the skin and system longer, has slower evaporation. Its antiviral nature helps block receptor sites of the herpes virus and assists immune function. Very recent advances in the scientific understanding of medicinal plants suggest a much broader use in the treatment for herpes infections than science thought possible just ten years ago.
These topical treatments can be beneficial for all cases of genital, oral, acute, chronic, or recurrent herpes infections. They also help to suppress excesses that take place in the urogenital tract or oral regions. To learn more, please go to http://www.forcesofnatureusa.com.
Following active infection herpes viruses establish a latent infection in sensory and autonomic ganglia of the nervous system. The double-stranded DNA of the virus is incorporated into the cell physiology by infection of the nucleus of a nerve's cell body. HSV latency is static (no virus is produced) and is controlled by a number of viral genes, including Latency Associated Transcript (LAT).
Many HSV infected people experience recurrence within the first year of infection. Prodrome precedes development of lesions. Prodromal symptoms include tingling, itching, and pain where lumbosacral nerves innervate the skin. Prodrome may occur as long as several days or as short as a few hours before lesions develop. Beginning antiviral treatment when prodrome is experienced can reduce the appearance and duration of lesions in some individuals.
During recurrence fewer lesions are likely to develop, lesions are less painful and heal faster (within 5-10 days without antiviral treatment) than those occurring during the primary infection. Subsequent outbreaks tend to be periodic or episodic, occurring on average four to five times a year when not using antiviral therapy.
The causes of reactivation are uncertain, but several potential triggers have been documented. A recent study showed that a protein VP16 plays a key role in reactivation of the dormant virus. Changes in the immune system during menstruation may play a role in HSV-1 reactivation.
Concurrent infections, such as viral upper respiratory tract infection or other febrile diseases, can cause outbreaks. Reactivation due to infection is the likely source of the historic terms cold sore and fever blister. Other identified triggers include: local injury to the face, lips, eyes, or mouth; trauma; surgery; radiotherapy; and exposure to wind, ultraviolet light, or sunlight.
The frequency and severity of recurrent outbreaks vary greatly between patients. Some individuals' outbreaks can be quite debilitating with large, painful lesions persisting for several weeks, while others will experience only minor itching or burning for a few days. An immunity to the virus is built over time.
Most infected individuals will experience fewer outbreaks and outbreak symptoms will often become less severe. After several years, some people will become perpetually asymptomatic and will no longer experience outbreaks, though they may still be contagious to others. Immunocompromised individuals may experience episodes that are longer, more frequent, and more severe.
Antiviral medication has been proven to shorten the frequency and duration of outbreaks. Outbreaks may occur at the original site of the infection or in proximity to nerve endings that reach out from the infected ganglia. In the case of a genital infection, sores can appear at the original site of infection or near the base of the spine, the buttocks, or the back of the thighs.
The herpes virus remains dormant in the ganglia, seemingly inactive, however once provoked/aggravated, the virus becomes symptomatic. It travels down the ganglia and forms blisters on the surface of the skin causing an outbreak, often reoccurring in the same location.
As the pathogenic factors are highly symptomatic and acute during an outbreak, a high intensity treatment should be used during this stage, whereas a gentler antiviral treatment should be used when the virus is dormant, to help act as suppressive therapy, which is why we developed HSVSuppressor-Rx.
HSVSuppressor-Rx is a softer antiviral treatment designed for daily application/ suppressive therapy. It stays in the skin and system longer, has slower evaporation. Its antiviral nature helps block receptor sites of the herpes virus and assists immune function. Very recent advances in the scientific understanding of medicinal plants suggest a much broader use in the treatment for herpes infections than science thought possible just ten years ago.
These topical treatments can be beneficial for all cases of genital, oral, acute, chronic, or recurrent herpes infections. They also help to suppress excesses that take place in the urogenital tract or oral regions. To learn more, please go to http://www.forcesofnatureusa.com.
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